Originally Posted by
burlyman30
I seem to remember Eric saying that the original AH was not actually androsterone, but epiandrosterone. I believe the the theory was that the a isomer had more anabolic effectiveness, so AH was reformulated in AH2 but the unexpected side effect was lethargy. AH3 added in the epiandrosterone in a ratio that was thought to combat this. I believe that the a isomer had an effect on the GABA receptors, causing the drowsiness.
Maybe h2s or one of the other former PP reps can add to this or correct me where I am off track.