Differences between R-DHEA and Super Epi DHEA
Hello Nostrum.
So I was hoping you can differentiate between these two compounds for me and others on the board who may need clarification.
They both convert to DHT, but there are obvious chemical differences between them.
Do they convert to different types of DHT?
I'm not 100% clear on what the differences are, but there are apparent differences in function and "feel"
Thanks!
Differences between R-DHEA and Super Epi DHEA
Quote:
Originally Posted by
TubZy
Here is another response from him, although it may not directly answer your question, which I may check up on as well. I honestly don't think it is a big issue when epiandro is paired with androsterone. I think it could be a bigger problem when epiandro is alone, again not confirmed though. So i'm not too worried about epi in regards to it being in the andro hard topical, IMO.
"I am not aware of any studies showing epiandro having better energetic or stimulating effects. If they can provide some studies that would be aswesome. There is a reason epiandro is so much cheaper than andro - it is less effective. Patrick Arnold said the same thing in one of his posts which you can probably find via Google - i.e. andro is where it's at and epiandro is just a poor substitute, which has been my experience as well. Not sure where their claim on using both being better is coming from. Again, epiandro is a 5b-isomer and may actually raise 5b-DHT instead of 5a-DHT. But if they have evidence I will obviously take it into account."
Yup....you're right....doesn't answer my question....lol
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Differences between R-DHEA and Super Epi DHEA
Quote:
Originally Posted by
TubZy
I don't want to give you the wrong answer haha. My guess is that since 5b-DHT is the "inactive androgen form" it could bind to the estrogen receptor beta much easier. I don't think he is saying "estrogenic" like gyno etc. (even though it is an agonist of ERb) just saying it is a much weaker version compared to androsterone. My post above mentions how much stronger 5a-DHT is compared to 5b-DHT in terms of binding (way way stronger). So 5b-DHT could just be a "by product" of the during the production of DHT which is why it is considered inactive, with weaker traits. It basically just assists in the conversion and deactivates afterwards.
I find it interesting that it would bind to the estrogen receptor at all.
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