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  1. #1
    Super Moderator Feedback Score 2 (100%) h2s's Avatar
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    "Old School" Steroid Profiles


    "Old School" Steroid Profiles
    The following profiles have been posted on many boards.
    I believe the original poster to be HawaiinPride of AAF. I am not sure if he has written the profile.
    If these profiles belong to you, please use the contact us feature.

  2. #2
    Super Moderator Feedback Score 2 (100%) h2s's Avatar
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    Turinabol

    Turinabol is an oral steroid which was developed during the early 1960's. Turinabol has a predominantly anabolic effect which is combined with a relatively low androgenic component. On a scale of 1 to 100 the androgenic effect is very low - only 6 - and the anabolic effect is 53. (In comparison: the androgenic effect of methandienone is 45 and its anabolic effect is 90.) Chlorodehydromethyltestosterone-turinabol is recommended in wasting diseases and HIV symptoms since it does not aromatize. Oral Turinabol is an oral steroid which was developed during the early 1960's.

    OT has a predominantly anabolic effect which is combined with a relatively low androgenic component. On a scale of 1 to 100 the androgenic effect is very low -only a 6- and the anabolic effect is 53. (In comparison: the androgenic effect of Dianabol is 45 and its anabolic effect is 90.) Oral-Turinabol thus has milligram for milli-gram a lower effect than Dianabol. It is therefore not a steroid that causes a rapid gain in strength, weight, and muscle mass. Rather, the achievable results manifest themselves in a solid muscle gain and, if taken over several weeks, also in a good strength gain. The athlete will certainly not get a puffy look as is the case with Test-osterone, Dianabol, and Anadrol 50. The maximum blood concen-tration of Oral-Turinabol when taking 10, 20 or 40 mg/day is 1.5 -3.5 or 4.5 times the endogenous testosterone concentration (also see Dianabol). This clearly shows that the effectiveness of this compound strongly depends on the dosage.

    0.4 x pound (body weight) x days = number of tablets to take overall during the interval of intake mg / tablet

    An athlete weighing 200 pounds would take only 4 tablets of 5 mg (20mg/day.) In our experience bodybuilders take 8-10 tablets of 5 mg, that is 40-50 mg/day. Many enthusiastically report good results with this dosage: one builds a solid muscle mass, the strength gain is worthwhile seeing, the water retention is very low, and the estrogen-caused side effects are rare. Not without good reason OT is also popular among powerlifters and weightlifters who appreciate these characteristics.

    Due to its characteristics OT is also a suitable steroid both for men and women in competitions. A usually very effective stack for male bodybuilders consists of 50 mg OT/day, 228 mg Parabolan/week, and 150 mg Winstrol Depot/week. Those who have brought their body fat content to a low level by dieting and/or by using fatburning substances (e.g. Clenbuterol, Ephedrine, Salbutamol, Cytomel, Triacana), will find that the above steroid combination will manifest itself in hard, sharply-defined but still dense and full muscles. No enlarged breasts, no estrogen surplus, and no watery, puffy-look-ing muscle system. If OT were available on the U.S. black market for steroids, bodybuilders, powerlifters, and weightlifters would go crazy for this East German anabolic.

    OT enjoys a great popularity since it is quickly broken down by the body and the metabolites are excreted relatively quickly through the urine. The often-posed question regarding how many days before a test OT can be taken in order to be "clean" is difficult to answer specifically or in general. We know from a reli-able source that athletes who only take OT as a steroid and who, in part, take dosages of 10- 15 tablets/day, have discontinued the com-pound exactly five days before a doping test and tested negative. These indications are supported by the fact that even positive urine analyses have rarely mentioned the names Oral-Turanabol or chlordehydromethyl-testosterone.

    The potential side effects of OT usually depend on the dosage level and are gender-specific. in women, depending on their predisposi-tion, the usual virilization symptoms occur and increase when dos-ages of more than 20 mg per day are taken over a prolonged time. In men the already discussed reduced testosterone production can rarely be avoided. Gynaecomastia occurs rarely with OT Since the response of the water and electrolyte household is not overly dis-tinct athletes only rarely report water retention and high blood pressure. Acne, gastrointestinal pain, and uncontrolled aggressive behavior are also the exception rather than the rule with OT An increased libido is reported in most cases by both sexes. Since the substance chlordehydromethyltestosterone is I 7-alpha alkylated the manufacturer in its package insert recommends that the liver func-tion be checked regularly since it can be negatively affected by high dosages and the risk of possible liver damage cannot be excluded. Thus OT is also a steroid that can be taken without interruption for long intervals. Studies of male athletes who over a period of six weeks were given 10 mg OT/day did not show any indications of health-threatening effects.

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    Anadrol
    Pharmaceutical Name: Oxymetholone
    Chemical structure: 17 beta-hydroxy-2-hydroxymethylene-17alpha-methyl-5 alpha-androstan-3-one
    Effective dose: 50-150 mg / day orally

    Oxymetholone is without a doubt the strongest and most visibly active steroid to date. Not only does it act very rapidly, it causes a virtual explosion of mass. Gains of up to 10 pounds in 2 weeks are not uncommon. This is largely due to a moderate to low androgenic effect combined with a high anabolic activity also mediated by non-AR mechanisms (mechanisms other than simply binding the androgen receptor). You can imagine that the gains made on oxymetholone aren't the leanest. You would note a drastic smoothing out of the muscle due to estrogen-related fat (lipolysis) and water retention. This lipolysis has been shown to be rather drastic. One study1 on long-term hemodialysis patients showed beyond a doubt the role that oxymetholone can play in causing hyperlipedemia. The fat deposition rate, post-hepatic (after processing by the liver), increased drastically in the oxymetholone group while numbers remained stable in the control group.

    It has been suggested that the estrogenic effects of oxymetholone may not be as much mediated by estrogen, as by oxymetholone itself activating the estrogen receptor. Because there is little to no aromatisation off oxymetholone, the possible progestational effect was examined first. Similar to that of nandrolone perhaps. But a study2 testing the progestational effects of oxymetholone and methandrostenolone against those of testosterone as well as nandrolone and its metabolites showed that the progestagenic activity of oxymetholone wasn't even in the neighbourhood of that of testosterone, let alone nandrolone. Ruling out the possibility of progestagenic activity and aromatisation, that only left oxymetholone engaging in a structure with the estrogen receptor itself. Since it has an A-ring similar to that of estradiol (the prime estrogen) so this would be the most logical explanation. Since progesterone acts as an estrogen agonist, it would require circulating estrogen to negotiate such levels of water build-up as oxymetholone causes, so it seemed like a far-fetched idea to begin with.

    The water component resulting from oxymetholone use is not be under-estimated either. The benefit of water retention is of course a lubrication of the joints, allowing the comfort of pain-free workouts even with extremely heavy weights, as well as the retention of more nutrients inside the cell, possibly leading to more permanent growth in muscle tissue. The downside to a massive water retention is that it gives you a rather puffed up look. A look not uncommon in off-season competitive bodybuilders and the heaviest classes of powerlifters. With the estrogen increase of course comes the increased risk of more side-effects such as gynocomastia (growth of breast tissue in men). Therefore its always advised that a cycle of oxymetholone is accompanied by the use of an anti-estrogen such as Nolvadex. Nolvadex, keeping in mind that aromatase enzyme is not involved, would be the wiser choice as it blocks the receptor for estrogen rather than the aromatase enzyme. Its wise to note as well that the gains from oxymetholone are largely mediated by estrogen, so reducing estrogen may reduce results as well.

    Because it is mild androgen as well as a potent estrogen, blood volume is increased. Androgens raise the red blood cells (although this has been shown to happen through a mechanism other than erythropoesis3) to improve aerobic performance while estrogens increase the white blood cells in an attempt to stimulate the immunity. Couple that increase in blood cells to an increase in water and you get a serious increase in blood volume. This effect has been known to result in magnificent pumps for the users of oxymetholone products. The synthesis of extra erythrocytes (Red blood cells) also increases stamina and performance (this effect is largely negated by the larger estrogenic component. Oxymetholone is not a good product for athletes). Together with the unbelievable strength effect of oxymetholone's water retention that makes for some incredible workouts. On a side note, these characteristics make for anadrol's popular use in treating anemia.

    The use of oxymetholone should be strict and brief. While it is no doubt the strongest steroid, quantitatively, its also by far the most hazardous steroid to your health. Apart from the great risk of common steroid-related side-effects (acne vulgaris, benign prostate hypertrophy, gynocomastia and androgenetic alopecia), it also has numerous other side-effects. Most notable is oxymetholone's hepatoxicity (damaging to the liver) : Its standard 17-alpha-alkylated as with most oral steroids, resulting in an inavoidable raise in liver transaminase enzyme counts. The most frequent of the hepatoxic effects is jaundice4 (yellow coloration of the skin) due to an oxymetholone induced increase in biliburine, but others include peliosis hepatis and formation of hepatic tumors (cancer). And that's not all. There is also a number of intrinsic side-effects noted with the use of this steroid. Headaches, stomach aches, nausea, vomiting, insomnia and diarrhea are among common afflictions associated with oxymetholone use.

    This is the reason why only strict doses of oxymetholone are used , often only 1-2 tabs of 50 mg. The general rule of thumb is to use 0.5 or 0.6 mg per pound of bodyweight, most likely putting you in the 100-150 mg range. Because of the negative effects on the liver, its often not used for more than a two or three weeks. The results are fast, but also fleeting and therapy is usually continued with another aromatizable compound, most likely a long acting testosterone like Sustanon or testosterone enanthate. The Anabolic Review also warns that under no circumstances should oxymetholone use exceed 6 weeks. When using oxymetholone, or any oral 17-alpha-alkylated steroid for that matter, one should always consult a physician on a frequent basis and get your liver values checked. Its not that oxymetholone is necessarily more toxic to the liver, but rather that much higher doses are needed than with other oral steroids, so the relative risk increases as well.

    Other notes I should mention about this compound are that oxymetholone's androgenic qualities are not linked to a 5-alpha reduced form. As a matter of fact it shows rather poor interaction with the 5AR enzyme, making it futile to treat a possible increase in hair loss with 5-alpha reductase-blocking products such as finasteride. Its androgenic component stems from the fact that oxymetholone is very much like Dihydrotestosterone were it not for the added 2-hydroxymethylene group. Since this group can be metabolically removed, that would leave methyl-DHT. A compound with a weaker affinity for the androgen receptor than straight DHT, but more active and with less affinity for the DHT-reducing enzyme 3beta hydroxysteroid dehydrogenase. Ultimately resulting in much stronger, instead of weaker androgenic effects than compounds that are actively 5-alpha reduced. This evens out largely, because the distribution is even across the body, where 5-alpha-reduction usually concentrates more potent androgenic forms in androgen responsive tissue such as skin and scalp.

    The effect on the blood pressure is rather drastic, so its recommend that you use a anti-hypertensive drug in conjunction, especially if you already have a fairly high blood pressure. Here too the care and control of a physician is advised. Because of the HPTA (hypothalamic-pituitary-testicular axis) suppressive nature, the use of Clomid or Nolvadex and HCG is advised as well towards the end of your oxymetholone use. Lastly, oxymetholone also has an ill effect on the glucose tolerance5, causing borderline diabetic situations. Something to be weary of if you yourself have been diagnosed with similar problems already. In conclusion one can safely state that the negative effects on the system associated with the use of this hormone are rather drastic and that the use is therefore not recommended for beginners, women or people who have pre-existing afflictions. Nonetheless Anadrol remains a popular steroid among experienced users to kick-start a steroid cycle because of its magnificent increases in strength and size. Most people who have used oxymetholone with great success have no problem calling it the strongest and most reliable steroid available today. A somewhat surprising remark however, since Methandrostenolone can produce similar results with half or a third of the doses normally used with oxymetholone and with less side-effects. So personally I would recommend methandrostenolone over oxymethelone, as its clearly stronger, milligram fro milligram. Oxymetholone remains a strong and favorable compound however, despite its side-effects. Its effects may also be slightly more explosive than those of methandrostenolone and therefore people seeking strength may give it an edge over the former.

  4. #4
    Super Moderator Feedback Score 2 (100%) h2s's Avatar
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    Andriol
    Pharmaceutical Name: Testosterone (as Undecanoate)
    Chemical structure: androsta-4-en-3-one,17b-ol
    Effective dose: 8-16 caps/day orally
    Available Doses: 40 mg/ml caps

    Andriol is a fairly recently developed steroid. A new attempt at making an orally available testosterone, the first since the very unpopular methyl-testosterone. The delivery system used for andriol is quite novel in itself and shows a lot of promise. If it weren't for a few quirks I'm sure this delivery method could have caught on fast. The crude methyl-testosterone was the first of many oral steroids delivered by way of a 17-alpha-methyl alteration to the base compound. Apart from changing the affinity for a lot a structures, making a steroid with completely different characteristics, the main problem here was that it invoked a level of hepatoxicity. Often minor, sometimes severe (anadrol, Halotestin). This meant that treatment could not be continued for extended periods of time in complete safety. The demand for an oral steroid that can be used for lengthy treatment has been high since the very beginning. First of all its never easy for a doctor to sell his patients on injection protocols (many fear or at least dislike needles) and for the doctor too it would be easier if the patient could take a pill than to have him come back every other week for an injection. So the pressure was on to create a steroid that wouldn't require a 17-alpha-methyl alteration.

    The answer was to seek a new way of delivery, that bypassed the liver, so that no alteration was needed to protect the steroid from being deactivated in the liver. That way was found in lymphatic absorption. As with many paths of uptake, this one too is very specific and limited. The lymphatic system is a series of heavily filtered channels intended for the resorption of water. When blood is delivered to a tissue through the arterial system, it is depleted of oxygen and nutrients, and then lead back to the heart by the venous system. Unfortunately only about 85% of the fluid is readily re-absorbed. That means 15% stays behind in the tissue and if that process where to continue day and night, we'd all swell up like Marshmallow man and explode in less than a week. That's why, inside tissues, there is another extended capillary system other than the cardiovascular one. The lymphatic system. This has the sole purpose of draining water from tissue. This is why it mostly only transports water. Its also heavily filtered by lymph nodes throughout the body that will remove almost everything, because the system is easily accessible and if not properly filtered a virus or cancer cell could easily spread throughout the body in this manner. But in the digestive tract it seems the lymph system makes an exception. Lymph fluid is usually clear (since its pure water), but in this area its troubled. That's because it appears to absorb oils and fats as well.

    Steroids are made from the prime storage of fat in the body, cholesterol. So there is a definite possibility here. And the lymphatic system, for 75-80%, empties itself in the major duct (ductus thoracicus), which in turn empties itself in the angulus venosus, where the vena jugularis interna (internal jugular vein) and the vena subclavia (vein below the collarbone) meet, right before they enter the heart through a common vein. That means, without having to pass the liver, these fats can be delivered straight to the heart. Now the question is, if indeed it was readily absorbed by the lymphatic system, why alter a steroid at all to survive the liver ? Obviously it doesn't get through to any great extent. That's because it absorbs only actual fats. This carrier therefore targets the solution of the steroid in an oil, so that it will be absorbed with the oil. It also seeks to make the compound more lipophillic so solution is more complete and permanent. As we also learned from injectable steroids, the way to make a compound more lipophillic is by attaching an ester. The longer the ester is, the more lipophillic it makes the steroid. In this case they opted for an undecanoate ester, which has a length of 11 carbons, the longest ester used to date.

    In this case we are talking about a testosterone undecanoate. It is dissolved in a type of sterile oil and then sealed inside a cap. As a whole, the dissolved steroid is then easily absorbed by the lymphatic system, prior to passing the liver, and delivered with ease to the heart where it is then sent out across the body. The system itself is ingenious and in theory perfect. Maximal delivery and no hepatoxicity. A potent steroid capable of being used for long treatments. However (I'm sure you saw that one coming) in practice things don't always turn out the way they appear in theory. In studies1 done on both men and women, andriol was shown to be a mild and inconsistent steroid at best. Mild is a problem that is easily solved with higher doses, but inconsistent is another story entirely. It seems that the amount that was delivered and the peak levels of testosterone in the blood as well as the length of activity, differed not only from person to person, but from day to day. That means a different person, from day to day, will get very varying levels of testosterone in the blood. And the differences were not minor.

    One subject may have a peak level of 5 ng/mmol while another can have in excess of 50 ng/mmol. What's worse, the same person may get these levels on different days. In terms of its anabolic (ie non-medical) use, that means doses of 8-16 caps per day are being used. That's more than most will inject per week of the shorter cypionate and enanthate esters. Normally 1 cap delivers 40 mg of testosterone undecanoate. An ester releases the steroid in the blood, leaving us with approximately 25 mg of testosterone per cap (it's a long and heavy ester). That's 200-400 mg per day being used, and andriol being as novel as it is, isn't cheap or easy to come by. That makes it, at best, just as uninteresting as methyl-testosterone.

    As far as the properties of this steroid go, like a propionate ester or a suspension injection, levels of testosterone, DHT and estrogen are easy to control, which makes this steroid a possibility for use during any time of the year, whether the athlete be cutting or bulking. The water retention is less notable than with longer esters, and if too high is easily controlled with Proviron or Nolvadex. Its pure testosterone, so if delivered in high enough doses, for reasons previously stated, it is of course a good mass builder with all the characteristics of testosterone. No more no less. It is of course a safe (to the liver), controllable oral steroid that can be used for extended periods of time, which does spark the interest of some, but anybody serious about gains will usually find andriol a very poor buy. Great invention for the medical world, but of little to no interest to the serious athlete.

    Andriol doesn't have that many uses. When utilized in doses of 8-16 caps per day are used and it can obviously be stacked with most any other steroid. Water retention problems that are common with testosterone are usually controllable enough to warrant use even during cutting phases, and even if not Proviron can be added to maximize its potential. The use of ancillaries is generally not required as its very mild to begin with and most problems can be solved by discontinuing use or lowering the dose. The usual anti-estrogens can be used, but generally with the cost of andriol for what little it does makes it less appealing to invest in the likes of Nolvadex or arimidex. Caps are best spread out throughout the day. Most oral steroids have a 17-alpha-methyl alteration that changes affinity and binding of the steroid, so that a single dose is usually enough. With andriol delivery is swift, peak doses high, but the steroid never outlasts its half-life of 3-5 hours. So it should be taken in three equal doses throughout the day, preferably with meals as lymphatic absorption is promoted in the presence of bile and other secretions in the GI tract.

  5. #5
    Super Moderator Feedback Score 2 (100%) h2s's Avatar
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    Deca-Durabolin
    Pharmaceutical Name: Nandrolone / Nor-testosterone (as undecanoate)
    Chemical structure: "19-Nor-4-androstene-3-one,17b-ol" or "4-Estren-17beta-ol-3-one"
    Effective dose: 200-800 mg / week

    The decanoate ester of nandrolone is generally referred to as Deca, stemming from the brand name Deca-Durabolin under which nandrolone was marketed by the Organon company. But as the reference list up above suggests there are many generic forms of this compound available. Nandrolone is perhaps the best marketed and easy to get steroid out there and it has always enjoyed an immense popularity. Its fairly accurate to state that safe for Dianabol, Deca is by far the most used steroid. The deca/d-bol stack, it is often suggested, is where the practice of stacking comes from. But what does it owe its popularity too ? Well, nandrolone has some unique qualities that make it unlike any other steroid known to man.

    Nandrolone is more commonly known as the base steroid 19Nor-testosterone. As this structure would indicate its like testosterone in appearance but for one small change : the absence of a carbon atom in the 19th position. This gives it a number of very distinct features. First of all it makes nandrolone a notably weaker agonist of the androgen receptor. That alone causes quite a reduction in the risk of androgenic side-effects. This is because it is the only steroid that is affected by the 5-alpha-reductase (5AR) enzyme in a way that makes it even less androgenic. Unlike testosterone which forms DHT (dihydrotestosterone) at the 5AR enzyme, a hormone 3-4 times as potent as an androgen receptor stimulator, nandrolone forms DHN (dihydronandrolone) a hormone that is even less suited than the already mild parent hormone for agonizing the androgen receptor. Those two features combined make nandrolone a very safe bet for people at risk for prostate hypertrophy, acne and aggravated male pattern hair loss. At the same time its estimated that nandrolone is 2.4 times as anabolic as testosterone1, on a gram for gram basis.

    Due to the many different ways that testosterone mediates anabolism, one has to take that statement with a serious grain of salt, but it does establish nandrolone as a potent muscle builder and performance enhancer with a comparatively safe character, at least androgenically speaking. This androgenic mildness is perhaps the greatest reason for its popularity. But due to the lack of immediate anabolic activity nandrolone is rarely used alone. Its the most known and sought after product for use as a base steroid, to use in conjunction with a more androgenic specimen to enhance the results without increasing androgenic side-effects to a serious degree.

    The ways in which nandrolone exerts its anabolic effects are two-fold. First of all it's a good mediator for nitrogen retention. When nitrogen retention is high, in essence it means that the cells are taking up more nitrogen than they are releasing. Why is this a good thing though? Well every amino acid has what is known as an amino-group, which contains nitrogen. When nitrogen is retained it means there is a high concentration of amino acids in a cell, which in turn positively affects the rate of protein synthesis. Since every tissue in the body is made from protein, including muscle, this means that muscle hypertrophy is facilitated. A second factor is through estrogen. While nandrolone's rate of aromatization is considerably smaller than that of testosterone, it does convert to a particularly powerful form of estrogeną. This has been noted to increase glycogen storage, growth hormone release and upgrade the androgen receptor in some tissues. In this case it also entails agonizing of aldosterone, but more on that later.

    It makes sense then that those particularly prone to the effects and side-effects of estrogen would take extra precaution. Blocking aromatase, considering the previous paragraph, would be a poor choice, but competitively inhibiting the estrogen receptor itself with clomiphene citrate (Clomid) or tamoxifen citrate (Nolvadex) might bring some relief since a large portion of progestagenic action is nullified if there is no circulating estrogen around, or if it is kept from being activated by the estrogen receptor. It is generally assumed that 1 mg of either every day for every 20 mg of nandrolone injected weekly is sufficient. Slightly higher doses, or the use of an aromatase inhibitor like cytadren can be stacked if nandrolone is used in conjunction with another aromatizing steroid. It has also been noted that the steroid stanozolol (Winstrol) may provide relief as it too binds to the progesterone receptor but remains unaltered by it. How strong of a competitor it is in such a case and what sort of doses would be needed are as much your guess as they are mine, so this may be non-issue. But it does bode well for the stacking of nandrolone with stanozolol in that you have nothing to lose and everything to gain.

    Another benefit of nandrolone use often reported is the pain-free workouts because nandrolone lubricates the joints. It stores a lot of water (as synovial fluid) in the joints, which eases the impact of the heavy weights handled by bodybuilders and weight lifters. One may wonder how nandrolone can do a better job at it than a steroid that aromatizes much stronger such as a testosterone ester, but its quite easily explained. One study at least goes to show that nandrolone metabolites are also aldosterone agonists6. Although we aren't entirely sure of the mechanism by which this occurs. But, while sparing you the details of this complex hormone, aldosterone has a strong function in the retention of sodium in the body. High sodium levels correlate with a high storage of water and that would explain the aforementioned effect. Of course one needs to note the implication of this of course: a bulkier frame and a certain loss of definition are not uncommon with nandrolone, perhaps more so than with testosterone.

    Nandrolone with a decanoate ester is fairly long acting (10 carbons) to begin with and if on top of that a lot of the drug can be de- and re-esterified that means the substance stays active in the body for quite a long time. This has resulted in positive drug tests for the hormone nandrolone and many of its metabolites, most notably 19-Norandrosterone up to 18 months after last use of the drug. While this is a fairly known fact, the recent number of athletes (including well known soccer stars) that have tested positive for nandrolone would indicate a lot of misinformation or plain lack of information in some circles. Positive tests, with reprimands, that could have easily been avoided. So anyone subject to any form of athletic drug test should refrain from using 19-Nortestosterone (nandrolone) or any of its metabolites, that includes nor-prohormones.

    Nandrolone stacks well with virtually anything. Due to its mildly aromatizing and its progestagenic activity its mostly used as a mass building compound by all but the monstrously huge. Because some water retention is a fact, one would not desire to include it in a cutting phase, especially if its one of your first cycles. Nandrolone is used in doses of 200-600 mg per week. 400 mg is the common recommendation for a somewhat experienced user, when used in conjunction with another product. Nandrolone as decanoate, as found in deca-durabolin, is a long acting ester of 10 carbons. That means 1 injection weekly will more than suffice as it has quite a long span of activity

    To this effect its preferably stacked with another aromatizing compound. In the first place a long acting testosterone like cypionate, enanthate or sustanon 250. For a beginner cycle, we want to note that the testosterone compound is the most active compound and its therefore more desirable to lower the dose of nandrolone rather than the dose of testosterone. Often beginners look to start at 400 mg of nandrolone and 250 mg of testosterone. A better suggestion would be 200 mg of nandrolone and 500 mg of testosterone. Then bump the nandrolone to 400 mg.

    It also makes a good match for doses of Anadrol or Dianabol, although neither compound can be used for the time-span of nandrolone decanoate due to liver toxicity. This isn't the case for a long-acting testosterone ester. Often nandrolone and test are stacked in conjunction with Anadrol or Dianabol for the first few weeks of a stack to boost gains and strength.

    A nandrolone stack accompanied by stanazolol (Winstrol/Stromba) makes sense as well, especially for those who are highly prone to gyno. It's commonly accepted that stanazolol can compete for the progesterone receptor, and since nandrolone can act as a progestin, this is a wise precaution. Progesterone agonizes estrogen and while nandrolone only aromatizes slightly and cases of gyno with moderate nandrolone use is rare, when stacking it with another aromatizable compound like Dianabol or testosterone, you may not want to take the chance. For secondary products one needn't consider an anti-aromatase like Cytadren since one cannot fully block all aromatase conversion and due to the enhanced estrogen activity as a result of progestagenic influence, it would serve little purpose. Using an estrogen-receptor antagonist, while not fool-proof obviously, may serve some benefit. Agonized or not, without binding to the receptor estrogen loses most of its influence. Using stanazolol and either clomid or Nolvadex during a stack with nandrolone is usually the best prescription. Post-cycle use of such substances to help HPTA recover faster and retain gains also comes highly recommended, and preferably for longer than you would with most stacks, since nandrolone stays active for a very long time

  6. #6
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    Equipoise
    Pharmaceutical Name: Boldenone (as undecylenate)
    Chemical structure: 1,4-androstadiene-3-one,17b-ol
    Effective dose: 300-600 mg/week

    For something that is generally injected into cows, horses and dogs boldenone is quite a popular and well-liked drug by most bodybuilders because of its unique make-up. It possesses several characteristics that aren't found in any other substance and its use is so varied its much desired year-round. Boldenone is a decent anabolic coupled with both a mild androgenic and a mild estrogenic effect. Sort of like a weak testosterone. In structure it doesn't differ all that much from testosterone, the main anomaly being a double bond in the one position as well as the 4 position. Its nonetheless quite good at promoting gains, but mostly through a combination of androgenic potential and other media than the androgen and estrogen receptors.

    The strange thing about its androgenic component is that it is mostly not mediated by a 5-alpha-reduced form, as is the case for most steroids. While it does indeed form a very potent 5AR form (dihydroboldenone, roughly 7 times as anabolic as testosterone1) its shows a very low affinity for the 5-alpha-reducatase enzyme2. This leads to the conclusion that a large part of the anabolic effect boldenone exerts is formed by the hormone itself binding to the androgen receptor. This could also be the reason its had such a successful run as a veterinary drug, because despite differences in the metabolism of species it has always produced extraordinary results.

    Like most anabolic steroids it increases muscle mass over time by increasing nitrogen retention and positively influencing protein synthesis or re-synthesis. An action that is not necessarily supported by an androgenic mediator as was shown with nandrolone. What boldenone has that other steroids don't is that it indirectly supplies the necessary means for that protein synthesis because it drastically increases the appetite. Thereby facilitating the high nutritional intake (especially protein wise) needed to book the best results when using anabolic androgenic steroids. Its more of a benefit than you think as a lot of people have theorized that it is this increase that is responsible for the great results booked when using boldenone. This theory may hold its own as there is indeed not much proof of the kind of anabolic activity with boldenone that would be responsible for the elicited effect.

    Its estrogenic activities are slight, but present. This has more of a positive than negative influence. The aromatisation of boldenone is too small to cause real problems and in normal doses (300-400 mg/week) problems such as gynocomastia and too much fat retention are unheard of. However small aromatisation is desirable as estrogen too mediates anabolic activity. It can be responsible for better glucose utilization3,4 (repleting lost glycogen stores after exercise) and stimulating increased growth hormone release5. But most notably estrogen is responsible for an upgrading of the androgen receptor6 allowing hormones that act on the androgen receptor to exert a larger anabolic effect. This is why hormones that are strong androgens but also aromatize heavily, like anadrol and testosterone, can put the most mass on your frame. In that aspect boldenone is perhaps the most suitable steroid because of its moderate estrogen levels that allow for the benefits, but not the side-effects of aromatization. And no doubt the perfect balance is partially responsible for stimulation of the appetite.

    For athletes of sports other than strength sports or bodybuilding will also note that boldenone is quite likely the most favorable steroid for them to use as it also stimulates the release of erythropoeitin in the kidneys. Erythropoeitin is a hormone known as EPO and heavily abused among endurance athletes because it signals the body to increase the production of red blood cells (erythrocytes). Red blood cells are the carrier of oxygen in the body, meaning that a higher maximal oxygen capacity can be obtained and better performance can be achieved over longer amounts of time before lactic acid is built up, which would in turn result in cramps and a cessation of the activity at that level. In short it improves your stamina. For bodybuilders this characteristic may be useful in promoting increased vascularity.

    In that aspect boldenone combined with a non-aromatizing steroid like Winstrol or Primobolan may be perfect to help you get cut and ripped while improving vascularity. The downside to that is that you really need to try hard to suppress the increased appetite. Which is why its probably a better idea to stack a somewhat larger dose of boldenone with a mass building drug like testosterone or anadrol to elicit major gains.

    The negative effects of boldenone are quite limited. In the normal doses of 300-400 mg a week estrogenic side-effects are almost never noted except in those who are very succeptible to estrogen. In terms of androgenic side-effects long-term use or very intense use of boldenone can cause slight virilizing effects such as acne and increased body-hair growth. Never really a problem for men, but women considering its use on account of its moderate androgenic qualities should be aware of this.

    As an undecylenate ester, boldenone needs only be injected every week (staying active well over 4 weeks), but because the preparations come in 25 mg/ml, users most often opt for 25-50 mg every day to every other day. A use of 300-400 mg per week seems to be the normal recommendation. Its not hepatoxic to any serious degree and can therefore be used for longer cycles. The appearance of underground forms of boldenone in higher concentrations (200 mg/ml) has made it easier to inject only once a week, which is to be preffered over the multiple dosings because it has a more even release and the cumulative effect shows much sooner. Speaking of cumulative effect, the best results with boldenone are seen when a user front-loads. Usually that means he will use a high doses of 600-800 mg/week for 2 weeks and then lower that dose to the normal 300-400 mg/week for the remaining 8-10 weeks.

    Boldenone is most often used for cutting. Its stacking partners for this purpose in particular are trenbolone, stanazolol and testosterone propionate. I'm no big fan of testosterone for cutting, although propionate is commonly used with great success by many users. Nonetheless I don't recommend test for cutting for beginners. Stanazolol is particularly useful in improving muscle hardness and strength while boldenone offers increased vascularity without overly aromatizing. The use of 50 mg of stanazolol every day, stacked with 300-400 mg per week of boldenone should serve the purpose of retaining gains and gaining increased definition and vascularity while shedding fat very well. Trenbolone would be a better match for those looking for moderate but very lean gains. Parabolan at 76 mg every other day for example will provide a decent increase in lean mass in combination with boldenone, without having to sacrifice shape or definition. Of course any combination of the above is an option as well. For example 300 mg of equipoise per week stacked with 76 mg of parabolan every other day and 50 mg of Winstrol every day, possibly with some test propionate at 50 mg a day.

    But though rarely mentioned, I personally find boldenone the better choice for bulking. Due to its effect on vascularity it is mostly used for cutting, but if you had a drug that increased your appetite like boldenone does, would you really use it to lose weight? It makes more sense to use it in a stack with a testosterone ester like enanthate or cypionate for good gains, instead of nandrolone. Sort of as a base. It aromatizes less than nandrolone and doesn't have that pesky progestagenic effect either, and because it increases appetite it would provide you with the means to an end in terms of gaining weight. 300-400 mg a week of boldenone with 500 mg of sustanon or 500 mg of testosterone enanthate would form an incredible stack. Even for those who prefer deca, adding a small amount of boldenone will go a long way in improving appetite. But boldenone is stronger than Deca, mg for mg, as well as safer and less suppressive.

    Boldenone makes a very poor match for nandrolone and methenolone though, since its very similar in action. The beauty of boldenone is that it can be an alternative for nandrolone when bulking due to its leaner results and more potent anabolic action, as well as an alternative for methenolone because while barely aromatizing its stronger than methenolone (Primobolan), gram for gram. The use of secondary drugs is rarely required. It doesn't aromatize at a great rate so the use of anti-aromatases is rarely implemented and the use of Nolva and clomid, during a cycle, is only necessary when stacked with aromatizing steroids like testosterone. Nolvadex or Clomid may have some use in restoring natural test post-cycle, because of the long-acting ester (11 carbons) and the mild estrogenic component. Normally 4 weeks of treatment is required, starting 1.5 to 2 weeks after the last shot.

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    Halotestin
    Pharmaceutical Name: Fluoxymesterone
    Chemical structure: 9-alpha-fluoro-11-beta-hydroxy-17-alpha-methyl-4-androstene-3-one,17b-ol
    Effective dose: 20-30 mg / day orally

    With the exception of perhaps anadrol, Halotestin is the single most dangerous steroid to use. Its liver toxicity is unrivaled and you wouldn't be the first person to end up in the hospital with jaundice and dangerously elevated liver values after a hefty cycle of fluoxymesterone. My question has often been simply "Why?". Fluoxymesterone has a low anabolic capacity. The results in mass would be small to non-existent. Qualitatively similar gains as one would book with trenbolone, but tren would go for equal or less money, deliver three times the gains and wouldn't be half as risky to use. Therefor the sole marked use of fluoxymesterone that is actually warranted is that by power- and weightlifters seeking to boost strength while remaining in a set weight class.

    In bodybuilding its used near the end of cutting cycles, since in people with an already low body-fat percentage it adds a distinct hardness and definition to the look, although, as stated, better and safer products will achieve similar effects. As with these alternatives fluoxymesterone has absolutely zero estrogenic activity and will thus not add water or fat to the frame in any way.

    While a definite increase in aggressiveness and a notable rise in erythrropoesis is noticed with the use of fluoxymesterone, it has been theorized that it actually has very moderate binding to the androgen receptor. Either that or it shows a higher affinity for other receptors. The enzyme aromatase comes to mind because of the effect it has, like a DHT compound would, on muscle hardness. The latter seems like a better explanation. On the one hand there is nothing that would immediately indicate it acting on the androgen receptor, on the other there is very good likeness to other steroids that are mostly AR-mediated. Its my best guess that not all has been said about fluoxymesterone. Its not a very interesting or grateful object of study however due to the high risk and low yield of this particular steroid.

    Athletes that may consider its use are endurance athletes that do not get drug tested (as it is quite easy to detect). The stimulating effect on erythropoesis (red blood cell production) and cell respiration, such an athlete would find a good use for the increase in aerobic capacity noticed for this, without adding unnecessary bodyweight to the frame he has to carry. In this aspect it may be good to note that a short cycle of Halotestin with a moderately long cycle of Equipoise may have some merit in this instance. Neither would increase water retention drastically, neither would give explosive gains. But both have positive effects on the VO2 max.

    In any case, and whatever the reason of use, 4 weeks is the best duration of use, 6 weeks at the most. As stated before, many athletes, having used fluoxymesterone while not under supervision of a physician, have ended up in the hospital with life-threatening conditions.

    Halotestin is taken in mild doses (10-20 mg) every day for short periods of time, 4 weeks, 6 weeks at the very most due to its high level of toxicity. The use of anti-estrogens is not necessary since fluoxymesterone does not aromatize at all. As secondary drugs one may want to consider blood pressure medication such as catepressan to avoid hypertensive conditions. What you will definitely need is a check of liver values on a regular basis if you want to play it safe. I don't normally recommend the use of liver-protectors during a cycle as enhances liver function breaks down a greater amount of your steroid, but in this case you ought to make an exception. Milk thistle, dessicated liver, vitamin B6 and such both during and after a cycle are highly advised. There is no need for clomid of Nolvadex use after a cycle to bring back natural test. Halotestin really only serves a purpose as a bodybuilding drug when the athlete is cutting. Probably in the late stages of a cutting cycle to promote muscle density and hardness, preserve muscle tissue and such. To that effect it may be good to use some Halotestin (20-30 mg/day) the last 4 weeks of a boldenone or methenolone cycle for example, or at the end of a stack with trenbolone. It may make a good stacking partner for stanazolol (Winstrol/Stromba) as well since they serve the same purpose. But frankly in all cases opting for a higher dose of the other drug may be a better choice, both in terms of gains and safety. Boldenone (Equipoise) being the one possible exception. Due to its toxicity Halotestin is not much sought out in stacks.

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    Methyltestosterone
    Pharmaceutical Name: Methyltestosterone
    Chemical structure: 17alpha-methyl-4-androstene-3-one,17b-ol
    Effective dose: 25-50 mg/day

    Methyltestosterone was, well, still is the worlds first oral steroid developed. Using the now infamous 17-alpha-methyl alteration to render the base hormone, testosterone, orally active. However, unlike the whole host of injectable testosterones, methyltest is a rather crude and not very well liked compound. Mostly due to this alteration. Methyltestosterone is to testosterone, what Dianabol (methandrostenolone) is to Equipoise (boldenone). On the one hand the 17-alpha-alkylation of the steroid gives it less affinity for the aromatase enzyme so less estrogen is formed, but as with Dianabol, the estrogen formed is 17-methyl-estradiol, which is much more potent. Just as we will notice serious bloat and water retention with Dianabol, we will see the same with methyltestosterone, but to a much greater degree, simply because the base structure has twice the tendency to aromatize. With this amount of estrogenic effects, gynocomastia is a very real threat and concomitant use of an anti-estrogen is strongly advised.

    The alteration also decreases the affinity for other structures. First and foremost the androgen receptor. This offers us few benefits. Due to the decreased androgenic activity the potency of methyltestosterone is weaker than that of testosterone, but even in terms of androgenic risk nothing is really gained. Testosterone being the prime androgen, even with this alteration risk of hair loss, acne, prostate hypertrophy and a whole host of other side-effects is never far away. Also, where Dianabol has little to no conversion to a more active androgen by way of the 5-alpha-reductase enzyme, methyl-testosterone still shows fair affinity for this particular enzyme and converts to the powerful 17-methyl-Dihydrotestosterone. These type of side-effects alone will turn most experienced users off of methyl-testosterone, at least when equally priced and more controllable injectable products are available. As with any potent androgen, some men may develop aggressive tendencies during its use.

    As with Dianabol, what we have on our hands here is a very potent mass builder and all in all an effective steroid when observed individually. 40-50 mg per day taken for just a few weeks can make drastic changes. But since many already find the bloat and fat gain of Dianabol a bit much to tolerate, this steroid is never in high demand. Dianabol is more available, provides extremely good results, is quite safe and comparatively cheap. So there is a multitude of reasons why methyltestosterone is rarely used. It seems, however, that it is making a re-introduction as a medical aid for oligospermic men, especially in the United States. One reason for this may in fact be the low demand for it on the black market, making more physicians comfortable in prescribing it due to a lowered chance of abuse.

    Lastly, as with all 17-alpha-alkylated steroids, we need to mention the risk for liver damage. A methyl-testosterone product used for extensive time periods can cause severe hepatoxicity, so use is best limited to 6, maximum 8 weeks on end followed by an off-period of equal length or longer.

    In conclusion, most will find methyl-testosterone to not be worth their while. The side-effects are ever present, and while they can easily be combated with a combination of arimidex and finasteride, it seems a bit idiotic to pay 15 or more dollars per day on ancillary drugs that will reduce the anabolic activity, while spending only 1-2 bucks at most on the steroid itself.

    Those still seeking to use methyltest will probably do so out of necessity and will not be stacking it with another anabolic/androgenic steroid. For such use 40-50 mg taken in a single daily dose upon waking, for a period no longer than 8 weeks would be ideal. Some may wish to use this steroid, like Dianabol, to kickstart a cycle and get results sooner at the beginning of a longer cycle of injectable testosterone, possibly stacked with another base compound such as boldenone or nandrolone. In that case 30 mg or so, again in a single morning dose, taken for the first 5-6 weeks of said cycle would provide the needed benefits. Since this is only useful in bulking stacks with aromatizable steroids, the resulting severity of side-effects will be grave. One needs to verify he is not at risk for hair loss or prostate hypertrophy first, and have ancillary drugs such as Nolvadex, arimidex and finasteride on hand to control the side-effects.

    In terms of ancillaries, If gynocomastia symptoms should appear, one should start the use of 20 mg of Nolvadex daily and start a cycle of 0.5 mg of anastrozole (arimidex) alongside it. After 3-4 days, the Nolva can be discontinued, but the anastrozole should be continued for a while longer. Some have asked me about the use of Proviron in this matter, but in my opinion one needs to realize how much DHT will be present with the use of this compound to begin with, it may do more harm than good to add more of it (Proviron being a 1-methyl-DHT). So granted, anastrozole is quite expensive, but needs to be given preference here. I don't normally approve of the use of finasteride, because DHT often offers a steroid user more benefits than problems (apart from those prone to hair loss) and the blocking thereof may reduce the results obtained, in this case, especially at the beginning of a longer injectable testosterone cycle, one may choose to look into its use. Natural testostosterone shutdown may be quite severe, so the use of HCG and Nolvadex or clomid post-cycle is virtually a must.

  9. #9
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    Miotolan
    Pharmaceutical Name: Furazabol
    Chemical structure: 17-alpha-methyl-5-alpha-androsta-2,3-furazan,17b-ol
    Effective dose: 20-50 mg/day

    Furazabol reminds us of Stanozolol (Winstrol) strucrurally. Its similar in appearance in that it's a DHT molecule with a 17-alpha-methyl group for oral availability, and has no 3-keto group, needed for androgenic binding. But instead of a 2,3-pyrazol group, furazabol has a 2,3-furazan group. The difference may not be all that big, both groups contain 2 nitrogen atoms and 2 double bonds and both are present instead of the 3-keto group. The advantage is that its not readily deactivated and therefore whatever influences it has, they are consistent. The downside is that the lack of a 3-keto group, which will impair its overall androgenic potency. So in that aspect again comparable to stanozolol. Anabolics 2002, without a doubt the best reference guide for steroids in print, lists Furazabol as extremely androgenic however, which is no doubt just an oversight. In nearly every way the behaviour of furazabol would be identical to that of Stanozolol.

    It's an obscure steroid, that's the least we can say. Its only manufactured in Japan and in tabs of 1 mg. Low availability makes the cost of this steroid rather high, and its not particularly easy to find. Perhaps a tad more potent than Stanozolol, the doses used lay in the same neighbourhood, 20-50 mg/day. The higher doses being the preference. The demand for it isn't very high either, because Winstrol/Stromba is a popular and cheap to come by. The only benefit of its obscurity is that noone will invest in faking it. So if you do come across Furazabol, you have pretty good odds that the stuff is legit.

    Now, the literature does not make a whole lot of mention of furazabol, but from what I was able to find1, it supports the weak nature of the steroid. In one case it was found that furazabol was a good treatment for hyperlipemia, and this without affecting proteinuria (the prevention of excretion of amino acids, where one would expect a steroid to increase proteinuria and not effect hyperlipemia). The low androgen binding may explain the lack of effect it had on proteinuria. The doses used were considerably high though, at least for furazabol. 1.1 mg/kg/day. That means a 200 lb bodybuilder would be using around 90-100 mg/day

    Furazabol can be considered a relatively light steroid therefore. It is not estrogenic in anyway, on account of its dihydro structure and its lack of estrogenic action and low androgenic binding make it have fairly little influence on the body's own testosterone production. Much like Winstrol (stanozolol) and Anavar (oxandrolone). In the long run suppression will occur of course, but because it occurs much slower a user will suffer less from testicular atrophy and therefore bounce back easier when a cycle is concluded. There is a slim chance of androgenic risk, as with Winstrol, but its not frequent or severe. So acne, increased body and facial hair and even an aggravation of male pattern hair loss can occur, but it's a lot less likely than with more androgenic specimen.

    Furazabol is a 17-alpha-alkylated steroid, and therefore has a level of hepatoxicity. In the interest of protecting your liver, you should not extend use beyond 6-8 weeks maximum. It's a mild steroid with no estrogenic activity, so logically its best used when cutting in stacks with Equipoise (boldenone undecylenate), Finaplix (trenbolone acetate) or Primobolan (methenolone enanthate) and the needed fat-burners of course. Unlike most steroids, this drug has a relatively short half-life2 however. It compensates with quite long activity (15-33% excretion of unchanged metabolites after 24 hours) so a single dose should be enough to get you through the day. But on account of the low half-life time, you may want to consider splitting doses in two each day. Because it doesn't aromatize and doesn't have a strong androgenic component, the use of ancillary drugs is limited. The use of Clomid or Nolvadex after a cycle is certainly advised, though the merit may be rather limited. There is no need for anti-estrogens or blood pressure medication during the cycle.

    Orabolin
    Pharmaceutical Name: ethylestrenol
    Chemical structure: 19-Nor-17alpha-pregn-4-en-17-ol
    Effective dose: 20-50 mg/day

    People who have been in this game for a long time, may remember this steroid as Maxibolin. Its most popular name. But to avoid all confusion, its now best referred to as Orabolin, because Maxibolan was taken from the market some time ago. Most experienced users will of course prefer not to remember Orabolin at all. It was a bit of a failure in all aspects.

    First of all it's a 19Nor-steroid, a derivative of nandrolone. I'm no big fan of 19Nor-steroids, apart from maybe trenbolone. The lack of the 19th carbon makes them re-esterify easily, particularly suppressive of natural testosterone and above all, lends them progestagenic activity, or if you will, the ability to worsen estrogenic side-effects by binding the progesterone receptor. The sole benefit of a 19nor compound is that it has very good androgen binding properties, giving it good enough anabolic effect, but is actually androgenically reduced in androgen responsive tissues like prostate and skin. This allowed users to book decent gains without overly having to fear acne, hair loss and prostate hypertrophy as they did with testosterone. For me that still doesn't warrant the use of nasty stuff like nandrolone or norethandrolone, for some it does. But I'm pretty sure all will be in agreement that this steroid is a waste. Its similar to norethandrolone, except it lacks a 3-keto group. This group is essential for binding the androgen receptor, and without it, its safe to assume that the anabolic activity of this steroid is less than weak. Studies1 actually seem to suggest that the only anabolic activity that ethylestronol does exert, is by making a 3-keto group and thus converting to norethandrolone.

    Norethandrolone (inviting you to read the profile on norethandrolone) wasn't much of a success either. It was designed to be an androgenically mild oral steroid, like an oral nandrolone (which is in essence what it was), but then Searle realized that apart from being androgenically mild it was relatively nasty and uncontrollable stuff (which is how I feel about most 19nor steroids, including nandrolone) and came out with Anavar instead, since it was better suited as a mild oral steroid. So even through conversion you don't get anything decent out of this product.

    The 17-alpha-ethyl group also lends it a certain liver toxicity, which doesn't allow for long use or high doses. And high doses are really what you need for any form of favorable effect. Women may somewhat appreciate this steroid in doses of 20-30 mg day, as its androgenically the mildest you'll ever come across, with very little virilizing symptoms. Although in any case, I would still recommend Anavar (oxandrolone) over ethylestrenol. Mainly due to its reputation, its become hard to find on the black market. Virtually extinct. I for one don't really care much.If you have ethylestrenol, my advice is toss it or sell it to your gullable friend. It's a waste. If you must use its, remember that the gains will be next to non-existent. Using 30-50 mg per day for 5-6 weeks on end, stacked with other products are the best way to go. Make it worth your while and add in some testosterone or boldenone for example. Aromatization is minimal, so the use of anti-estrogens will not be needed during the cycle, but because 19Nor steroids are nasty, suppressive stuff, you would do wise to run HCG and Nolvadex or Clomid once the cycle is over

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    Primobolan
    Pharmaceutical Name: Methenolone (orally as acetate, injections as enanthate)
    Chemical structure: 17 beta-hydroxy-1-methyl-5 alpha-androst-1-en-3-one
    Effective dose: 200-300 mg/week injections or 50-150 mg/day orally

    Primobolan is a well-known and popular steroid as well. Like nandrolone it's most often used as a base compound for stacking with other steroids. Methenolone however, is a DHT-based steroid (actually, DHB or dihydroboldenone, the 5-alpha reduced of the milder boldenon). Meaning when it interacts with the aromatase enzyme it does not form estrogens at all. That makes it ideal for use when cutting when excess estrogen is best avoided because of its retentive effects on water and fat. Methenolone is mostly only used in such instances, or by people who are very succeptible to estrogenic side-effects, because the anabolic activity of methenolone is slightly lower than that of nandrolone, quite likely BECAUSE it is non-estrogenic.

    Because it is a widely available steroid its often used as a replacement for nandrolone or boldenone to those who have no access to Deca-Durabolin or Laurabolin or Equipoise. When stacked with a heavy mass steroid like testosterone and/or methandrostenolone it can deliver almost similar gains. Those seeking to cut will most likely be very pleased stacking it with drostanolone, stanozolol or trenbolone. Women and beginners also stack methenolone WITH nandrolone because this gives a mildly anabolic stack that is generally regarded as one of the safer stacks around in an androgenic perspective. But alas, with the nandrolone, also a very suppressive stack.

    Methenolone is available as an injection or as an oral. The injection is naturally regarded as better. Its an enanthate ester which is quite long-acting and only needs to be injected once a week in doses of 300-600 mg. Because it by-passes hepatic breakdown on the first pass, it also has a higher survival rate. The orals are a lot less handy, but often preferred by bodybuilders who are afraid of needles or who are already taking one or more injectable compounds. The tabs are in a short-lived acetate form, meaning that doses of 100-150 mg per day are needed, split over 2 or 3 doses, making the tabs quite inconvenient for use. The reason doses need to be split up, unlike most oral steroids, is because Methenolone is not 17-alpha-alkylated, but 1-methylated for oral bio-availability. This reduces the liver stress, but also the availability, hence the multiple and high doses needed daily.

    Like nandrolone, methenolone is very mild on the system. Probably the reason why both are strongly favored as base compounds in stacks. Methenolone has no estrogenic side-effects whatsoever, on account of its structure. Its effects on the cholesterol levels are barely noticeable. In doses of 200 mg or less (injectable) blood pressure is rarely, if at all, altered. As for hepatoxicity, long-term use will of course increase liver values but gradually and only slightly. The injections of course, since they only pass the liver once, have roughly half the liver-toxic effects of the tabs. The low liver-toxicity is accounted for that the bio-availability of methenolone is carried by a 1-methyl-group, which lessens the need for a carrier attachment such as a 17-alpha-akylated group, the main culprit in steroid-related liver afflictions.

    The strangest thing however, taking into account that Primo is still a DHT (or rather DHB) derivative, is that it is quite easy on the system androgenically as well. Women use methenolone often, usually the tabs, and find little virilisation symptoms in short term use of methenolone. Long-term use may induce some acne and a deepening of the voice however. Methenolone is also not overly suppressive of the HPT axis (endocrinal axis for the production of natural testosterone). These are both the result of DHB's 1,2-double bond, which, analog to the parent structure boldenone, reduces the androgenic binding by 50% as opposed to DHT.

    For athletes who wish to maintain a "natural" status in competition, the tablets are quite well-suited as detection chances for the acetate-form are quite slim. However tests have improved and quite a number of metabolites1 of methenolone can be detected with a simple urine sample. But an English study documented that there is a liability in eating methenolone contaminated meats2, which could provide a possible defense if found out. One could always claim they ate the meat of a chicken or cow injected with methenolone since the test concluded eating such meat does not improve performance, but can deliver positive tests for several methenolone metabolites almost 24 hours after ingestion. That's for those of you seeking a viable defense against a possible methenolone-positive.

    Methenolone comes in orals and injectables. The injectables are to be preferred as they can be used for quite some time and only require injecting once a week. The orals are taking every day, or multiple times a day. An oral passes through the liver twice. An injectable only once. The injectable is more effective since less is broken down.

    Methenolone is not used all that often by experienced users. It makes a good product as an alternative to Deca or EQ in a cutting stack, because it has similar properties but does not aromatize and does not have progestagenic activity. But those at least slightly versed will prefer boldenone over methenolone as its more potent gram for gram. Its quite mild, so its not as prone to cause your standard side-effects. This too makes it quite popular with beginners. Methenolone was quite popular during the 70's in stacks with Methandrostenolone. Some of the all-time greats of bodybuilding were quite fond of this stack. The common use is similar to that of Nandrolone. 300-400 mg a week, in conjunction with other steroids mostly. Some attempt to make up for the lack of potency switching from nandrolone or boldenone to methenolone by using higher doses, in the neighbourhood of 600-800 mg a week. At that point I feel it would be cheaper to opt for boldenone at 300-400 mg a week though. Methenolone makes a poor stacking partner in mass stacks as both Deca and EQ provide better results while they are qualitatively similar. There is a slight merit in stacking Methenolone with boldenone, because apart from its 1-methyl group, methenolone is basically DHB, the 5-alpha-reduced form of boldenone. But since boldenone itself has very low affinity for 5-alpha-reduction, it should have a good synergistic effect stacking the two at 300 mg/week each.

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